PsBattle: Cat looking into lampSubmitted by happysheeple3 t3_1235950 in photoshopbattles
happysheeple3
PsBattle: Mike McCarthy Stiff-Arms a CameramanSubmitted by happysheeple3 t3_10jeau4 in photoshopbattles
happysheeple3 t1_isvq1ib wrote
Reply to comment by greyrobot6 in Severe COVID-19 may increase the risk for schizophrenia by chrisdh79
I take it before bed and I don't ever feel high. It only takes 25mL of the full spectrum tincture to get results. Music will sound a little nicer, but no head rush unless I overdo it with like 75mL.
happysheeple3 t1_isus0ew wrote
Reply to comment by greyrobot6 in Severe COVID-19 may increase the risk for schizophrenia by chrisdh79
I'm so happy to hear a Dr. that's willing to say that! So many of them mindlessly believe what they're taught in school which in many cases is antiquated or outright false information. It warms my heart to hear that!
If you don't partake in marijuana already, try a THC tincture. Make sure it's full spectrum and isn't flavored. I had inflammation which I saw great improvement in when I started. CBD can work too, but it isn't as strong and the effects don't last as long, at least in my personal experience.
If you're easily addicted to things, stick with cbd.
happysheeple3 t1_isu474m wrote
Reply to comment by Dawrin in Severe COVID-19 may increase the risk for schizophrenia by chrisdh79
I hope we get one that explores a possible risk stratification for serious outcomes from covid-19 with varying intakes of added sugars (sucrose, fructose and high fructose corn syrup)
The average American consumes around 57 pounds of sweeteners per year. Added sugars including sucralose and aspartame have been found to be Pro-inflammatory and could very well take a large part of the blame for the covid-19 "cytokine storms" described in the literature.
>Fructose pathway involved in oxidative stress and inflammation. In the hepatocyte, fructose sugar is rapidly metabolized by KHK, depleting ATP and resulting in uric acid accumulation. Uric acid activates NADPH oxidase with production of ROS, which activates mitochondrial ROS production and stimulates UPR in ER. ER stress may also activate mitochondrial ROS production and vice versa. Moreover, ER stress contributes to progression of hepatic steatosis activating lipogenic genes via PERK, ATF4, and IRE1 and inducing SREBP-1c that increases the activity of ACC and FAS. As a response of excessive oxidative stress, inflammatory and apoptotic pathways are activated through JNK, NFkB, and CHOP, resulting in an increased production of cytokines (IL1ß, TNFa, IL19), which plays an important role in the development of NASH.
happysheeple3 t1_itpakvu wrote
Reply to [Image] Stop Treating Yourself Like an After Thought. by poptartjake
Eat the delicious food all you want. Just don't ask me to foot the bill for your cholesterol and blood pressure meds.